Rapid initiation
Citrated Rapid TEG — tissue factor plus kaolin activation. Fastest channel to results in active hemorrhage; CRT.MA gives an early read on clot strength when waiting on conventional labs costs blood.
Nurse Anesthesiology / Trauma & Coagulation
A nurse anesthesiology resident at the University of Arizona, focused on trauma resuscitation and TEG 6s–guided hemostatic management.
About
I am a Doctor of Nursing Practice candidate in nurse anesthesiology at the University of Arizona, with clinical rotations across Phoenix’s Level I trauma centers — Banner University, Banner Desert, and Valleywise — covering general and trauma surgery, orthopedic trauma, pediatrics, obstetrics, and burns.
Before training, I spent two years in the Trauma / Surgical ICU at Renown in Reno, caring for high-acuity patients drawn from over 80,000 square miles of catchment. That ground — complex resuscitation, vasoactive titration, invasive monitoring — shapes how I think at the head of the bed today.
My clinical and scholarly focus sits at the intersection of trauma anesthesiology and viscoelastic coagulation testing — specifically TEG 6s with the Global Hemostasis with Lysis cartridge — to drive goal-directed resuscitation when minutes and products both matter.
Clinical & Research Focus
The Global Hemostasis with Lysis cartridge runs three assays in parallel — CK, CRT, and CFF — producing a profile of clot initiation, clot strength, fibrinogen contribution, and fibrinolysis from a single citrated sample. In trauma, that distinction reshapes the resuscitation.
Hover a parameter below to highlight its channel on the tracing.
Citrated Rapid TEG — tissue factor plus kaolin activation. Fastest channel to results in active hemorrhage; CRT.MA gives an early read on clot strength when waiting on conventional labs costs blood.
Citrated Kaolin — the workhorse channel. CK.R reflects clot initiation and points us toward plasma rather than reflexive platelet transfusion; CK.MA is the integrated read on overall clot strength.
Citrated Functional Fibrinogen — platelets are inhibited so the MA reflects fibrinogen alone. FFMA reframes cryoprecipitate or concentrate as a goal-directed decision rather than empiric replacement.
Catching trauma-induced hyperfibrinolysis early changes mortality. CK.LY30 operationalizes the TXA decision in real time — a small number with outsized weight for the patient on the table.
“Goal-directed resuscitation is not just about giving less product. It is about giving the right product at the right moment — and being able to explain why at the end of the case.”
Publications & Media
Peer-reviewed work, podcast appearances, talks, and projects in motion. This page grows with the practice.
Integrative review synthesizing the current evidence on TEG 6s adoption in trauma anesthesia — cartridge selection, parameter interpretation, and integration with massive transfusion protocols.
Read the articleConversations on viscoelastic testing in trauma resuscitation and the practical realities of implementing TEG 6s at the bedside.
Episodes will appear herePosters, lectures, and workshop sessions on TEG 6s, goal-directed transfusion, and trauma anesthesiology.
Coming soonQuality improvement and translational projects from the trauma rotations — updates appear here as they progress to publication.
More to comeCurriculum Vitae
Education, training, certifications, and ongoing scholarly work.
A current PDF of my curriculum vitae — updated regularly with clinical experience, certifications, and research activity.
Contact
Clinical opportunities, collaboration, or a question about TEG 6s in trauma — send a note.